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A showed only low sequence similarity to mouse proteins (BLASTP E-Yadetie et al. BMC Genomics 2012, 13:55 http://www.biomedcentral.com/1471-2164/13/Page 6 ofTable 1 Comparison of numbers of selected defensome genes in genomes of Oikopleura and other organismsGene AhR signaling CYP1 genes AhR AhRR ARNT Nuclear receptors NR1I (SXR/CAR/ VDR) NR1H (LXR/FXR) NR1C (PPAR) Nrf2-signaling Nrf2 Keap1 MafKaO
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Ate xenobiotic receptors. No strong candidate for PPAR (NR1C) was found in Oikopleura (our unpublished data).We further explored the Oikopleura genome for transcription factors involved in oxidative stress response, particularly the Nrf2-complex and found homologs of Nrf2, kelch-like ECH-associated protein 1 (Keap1), and small Maf proteins (Table 1, Additional file 1, Figure S2). Phylogenetic comp
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Er This property is particularly . suitable for vaccine design. There isEr This property is particularly . suitable for vaccine design. There is no limitation to the size of peptide displayed, given the ability of the E2 CD to naturally present 60 lipoyl domains plus 60 copies of the E1 (150 kDa) or E3 (100 kDa) enzymes. [61] Domingo et al demonstrated that a green fluorescentWJV|www.wjgnet.
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S low-cost, nonreplicating, non-integrating, non-pathogenic E2 vaccines could be designed andS low-cost, nonreplicating, non-integrating, non-pathogenic E2 vaccines could be designed and combined with other approaches to advance the field of vaccinology.CONCLUSIONVaccines play a pivotal role in host protection against infectious diseases and have significantly reduced mortality worldwide. Ho
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Set of 67 manually assigned backbone NOEs, and RDCs, we obtain a dimer structure that is within 2.6 ?backbone rmsd from the homologous structure (Supporting Information Figure 2). Furthermore, the structures are very similar in fold to the structures obtained using conventional structure calculations and a full set of NOEs. Good agreement with the RDCs is indicated by a Q-factor of 0.32 (see Mater
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D in the list of genes used for pathway analysis (see Methods).Pathways affected by BaPcancer, small cell lung cancer and nitrogen metabolism (Table 2). Pathways related to cancer reflect the mouse annotations, but some of the genes in the pathways appear to be involved in conserved cellular processes such as DNA damage and oxidative stress responses. MetaCore enrichment analyses showed most enric
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Ere phenotype in these mice is due to the existence of a compensatory loricrin back-up system. Increased expression of small proline rich proteins has been observed in the CE of Lor2/2 mice. Interestingly expression of other CE components such as involucrin, filaggrin and Cytokeratin 10 (K10) were similar in Lor2/2 and wild-type mice [21]. Two S. aureus surface proteins, clumping factor B (ClfB) a
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Accharide (LPS) component of the outer cell membrane. Such a toxicAccharide (LPS) component of the outer cell membrane. Such a toxic component triggers secretion of pro-inflammatory cytokines, and it often requires extensive and expensive removal during protein purification, thus affecting the final yield. It was shown that solubility and stability of recombinant E2 scaffolds that precipitat

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